By Icon Health Publications
This can be a 3-in-1 reference ebook. It offers an entire scientific dictionary masking hundreds and hundreds of phrases and expressions on the subject of acute myeloid leukemia. It additionally supplies vast lists of bibliographic citations. eventually, it offers details to clients on find out how to replace their wisdom utilizing numerous web assets. The ebook is designed for physicians, scientific scholars getting ready for Board examinations, clinical researchers, and sufferers who are looking to get to grips with learn devoted to acute myeloid leukemia. in case your time is efficacious, this ebook is for you. First, you won't waste time looking the web whereas lacking loads of appropriate info. moment, the ebook additionally saves you time indexing and defining entries. eventually, you won't waste money and time printing hundreds and hundreds of web content.
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Additional resources for Acute Myeloid Leukemia - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References
It is claimed that each case-control study has adequate statistical power to address the hypotheses being explored. ; Fred Hutchinson Cancer Research Center Box 19024, 1100 Fairview Ave N Seattle, Wa 98109 Timing: Fiscal Year 2002; Project Start 01-JUL-2002; Project End 30-JUN-2007 Summary: (provided by applicant): Dr. Stirewalt's long-term objective is to develop treatments for patients with AML that will reduce morbidity and improve survival. Over the next five years, Dr. Stirewalt will perform pre- clinical and clinical studies that examine the effects of FLT3 mutations in AML.
The MRD assays for FLT3 ITDs will be performed in all FLT3 positive patients at specific time points during their treatment and results will be correlated with clinical outcomes. We hypothesize that MRD assays using FLT3 ITDs will be predictive of clinical relapse. Specific Aim III: Examine the molecular effectors involved in the activation of mutated FLT3 receptors and develop techniques to counteract the effects of mutated FLT receptors. Data from murine cells with transfected FLT3 ITDs suggest that FLT3 ITDs result in activation of the FL T3 receptors that can be inhibited by nonspecific tyrosine kinase inhibitors.
However, there still remains a sizable fraction of cases for which molecular predictors of prognosis are not clinically useful, because the malignant clone either lacks such an abnormality or because the abnormality is too infrequent to correlate with clinical outcome. The CALGB is a leader in using cytogenetic and molecular markers to stratify patients based on the risk of failure with standard treatment, and the identification of such indicators has become central to assignment of the appropriate therapy.